Control of tachyarrhythmias associated with Wolff-Parkinson-White syndrome by amiodarone hydrochloride
Identifieur interne : 000152 ( Main/Corpus ); précédent : 000151; suivant : 000153Control of tachyarrhythmias associated with Wolff-Parkinson-White syndrome by amiodarone hydrochloride
Auteurs : Mauricio B. Rosenbaum ; Pablo A. Chiale ; David Ryba ; Marcelo V. ElizariSource :
- The American Journal of Cardiology [ 0002-9149 ] ; 1974.
Abstract
Amiodarone hydrochloride proved to be highly effective in preventing and treating arrhythmias of the Wolff-Parkinson-White (WPW) syndrome in 11 patients with WPW conduction and recurrent tachyarrhythmias. Paroxysmal supraventricular tachycardia (six patients), atrial fibrillation (four patients) and atrial flutter (one patient) were the most significant arrhythmias. In most patients the arrhythmia was seriously disabling because of the extremely rapid ventricular rate, adverse hemodynamic consequences and frequent recurrence and long duration of the episodes. Other known antiarrhythmic agents were ineffective. In all 11 patients amiodarone, in doses of 300 to 600 mg daily, totally, easily and safety controlled the arrhythmias for periods of 2 to 8 months. The drug was fully effective after an average of 7 days of treatment. Tolerance to amiodarone was excellent. The occurrence of corneal microdeposits of the drug was the only Important undesirable effect, but subjective ocular disturbances were not noted. The microdeposits are reversible, and can be avoided by discontinuing the drug for 7 days every 1 to 2 months. Amiodarone apparently causes a significant prolongation of refractoriness in the normal (A-V node and His-Purkinje system) as well as in the anomalous pathway, thus creating favorable conditions for prevention and Interruption of any reentry mechanism requiring participation of both pathways.
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DOI: 10.1016/0002-9149(74)90200-8
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Chiale</name><affiliation><mods:affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</mods:affiliation></affiliation></author><author><name sortKey="Ryba, David" sort="Ryba, David" uniqKey="Ryba D" first="David" last="Ryba">David Ryba</name><affiliation><mods:affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</mods:affiliation></affiliation></author><author><name sortKey="Elizari, Marcelo V" sort="Elizari, Marcelo V" uniqKey="Elizari M" first="Marcelo V." last="Elizari">Marcelo V. 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Rosenbaum</name><affiliation><mods:affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</mods:affiliation></affiliation></author><author><name sortKey="Chiale, Pablo A" sort="Chiale, Pablo A" uniqKey="Chiale P" first="Pablo A." last="Chiale">Pablo A. Chiale</name><affiliation><mods:affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</mods:affiliation></affiliation></author><author><name sortKey="Ryba, David" sort="Ryba, David" uniqKey="Ryba D" first="David" last="Ryba">David Ryba</name><affiliation><mods:affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</mods:affiliation></affiliation></author><author><name sortKey="Elizari, Marcelo V" sort="Elizari, Marcelo V" uniqKey="Elizari M" first="Marcelo V." last="Elizari">Marcelo V. Elizari</name><affiliation><mods:affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">The American Journal of Cardiology</title><title level="j" type="abbrev">AJC</title><idno type="ISSN">0002-9149</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1974">1974</date><biblScope unit="volume">34</biblScope><biblScope unit="issue">2</biblScope><biblScope unit="page" from="215">215</biblScope><biblScope unit="page" to="223">223</biblScope></imprint><idno type="ISSN">0002-9149</idno></series><idno type="istex">966F15CA02BF8F246CCD9A5BAF00362631A1117D</idno><idno type="DOI">10.1016/0002-9149(74)90200-8</idno><idno type="PII">0002-9149(74)90200-8</idno><idno type="ArticleID">74902008</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0002-9149</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Amiodarone hydrochloride proved to be highly effective in preventing and treating arrhythmias of the Wolff-Parkinson-White (WPW) syndrome in 11 patients with WPW conduction and recurrent tachyarrhythmias. Paroxysmal supraventricular tachycardia (six patients), atrial fibrillation (four patients) and atrial flutter (one patient) were the most significant arrhythmias. In most patients the arrhythmia was seriously disabling because of the extremely rapid ventricular rate, adverse hemodynamic consequences and frequent recurrence and long duration of the episodes. Other known antiarrhythmic agents were ineffective. In all 11 patients amiodarone, in doses of 300 to 600 mg daily, totally, easily and safety controlled the arrhythmias for periods of 2 to 8 months. The drug was fully effective after an average of 7 days of treatment. Tolerance to amiodarone was excellent. The occurrence of corneal microdeposits of the drug was the only Important undesirable effect, but subjective ocular disturbances were not noted. The microdeposits are reversible, and can be avoided by discontinuing the drug for 7 days every 1 to 2 months. Amiodarone apparently causes a significant prolongation of refractoriness in the normal (A-V node and His-Purkinje system) as well as in the anomalous pathway, thus creating favorable conditions for prevention and Interruption of any reentry mechanism requiring participation of both pathways.</div></front></TEI><istex><corpusName>elsevier</corpusName><author><json:item><name>Mauricio B. Rosenbaum MD</name><affiliations><json:string>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</json:string></affiliations></json:item><json:item><name>Pablo A. Chiale MD</name><affiliations><json:string>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</json:string></affiliations></json:item><json:item><name>David Ryba MD</name><affiliations><json:string>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</json:string></affiliations></json:item><json:item><name>Marcelo V. Elizari MD, FACC</name><affiliations><json:string>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</json:string></affiliations></json:item></author><articleId><json:string>74902008</json:string></articleId><language><json:string>eng</json:string></language><abstract>Amiodarone hydrochloride proved to be highly effective in preventing and treating arrhythmias of the Wolff-Parkinson-White (WPW) syndrome in 11 patients with WPW conduction and recurrent tachyarrhythmias. Paroxysmal supraventricular tachycardia (six patients), atrial fibrillation (four patients) and atrial flutter (one patient) were the most significant arrhythmias. In most patients the arrhythmia was seriously disabling because of the extremely rapid ventricular rate, adverse hemodynamic consequences and frequent recurrence and long duration of the episodes. Other known antiarrhythmic agents were ineffective. In all 11 patients amiodarone, in doses of 300 to 600 mg daily, totally, easily and safety controlled the arrhythmias for periods of 2 to 8 months. The drug was fully effective after an average of 7 days of treatment. Tolerance to amiodarone was excellent. The occurrence of corneal microdeposits of the drug was the only Important undesirable effect, but subjective ocular disturbances were not noted. The microdeposits are reversible, and can be avoided by discontinuing the drug for 7 days every 1 to 2 months. 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Rosenbaum, MD, Rivadavia 3820, P.B., A, Buenos Aires, Argentina.</note><affiliation>Address for reprints: Mauricio B. Rosenbaum, MD, Rivadavia 3820, P.B., A, Buenos Aires, Argentina.</affiliation><affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</affiliation></author><author><persName><forename type="first">Pablo A.</forename><surname>Chiale</surname></persName><roleName type="degree">MD</roleName><affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</affiliation></author><author><persName><forename type="first">David</forename><surname>Ryba</surname></persName><roleName type="degree">MD</roleName><affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</affiliation></author><author><persName><forename type="first">Marcelo V.</forename><surname>Elizari</surname></persName><roleName type="degree">MD, FACC</roleName><affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</affiliation></author></analytic><monogr><title level="j">The American Journal of Cardiology</title><title level="j" type="abbrev">AJC</title><idno type="pISSN">0002-9149</idno><idno type="PII">S0002-9149(00)X0400-6</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1974"></date><biblScope unit="volume">34</biblScope><biblScope unit="issue">2</biblScope><biblScope unit="page" from="215">215</biblScope><biblScope unit="page" to="223">223</biblScope></imprint></monogr><idno type="istex">966F15CA02BF8F246CCD9A5BAF00362631A1117D</idno><idno type="DOI">10.1016/0002-9149(74)90200-8</idno><idno type="PII">0002-9149(74)90200-8</idno><idno type="ArticleID">74902008</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1974</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>Amiodarone hydrochloride proved to be highly effective in preventing and treating arrhythmias of the Wolff-Parkinson-White (WPW) syndrome in 11 patients with WPW conduction and recurrent tachyarrhythmias. Paroxysmal supraventricular tachycardia (six patients), atrial fibrillation (four patients) and atrial flutter (one patient) were the most significant arrhythmias. In most patients the arrhythmia was seriously disabling because of the extremely rapid ventricular rate, adverse hemodynamic consequences and frequent recurrence and long duration of the episodes. Other known antiarrhythmic agents were ineffective. In all 11 patients amiodarone, in doses of 300 to 600 mg daily, totally, easily and safety controlled the arrhythmias for periods of 2 to 8 months. The drug was fully effective after an average of 7 days of treatment. Tolerance to amiodarone was excellent. The occurrence of corneal microdeposits of the drug was the only Important undesirable effect, but subjective ocular disturbances were not noted. The microdeposits are reversible, and can be avoided by discontinuing the drug for 7 days every 1 to 2 months. Amiodarone apparently causes a significant prolongation of refractoriness in the normal (A-V node and His-Purkinje system) as well as in the anomalous pathway, thus creating favorable conditions for prevention and Interruption of any reentry mechanism requiring participation of both pathways.</p></abstract></profileDesc><revisionDesc><change when="1974-01-09">Registration</change><change when="1974">Published</change></revisionDesc></teiHeader></istex:fulltextTEI></fulltext><metadata><istex:metadataXml wicri:clean="Elsevier, elements deleted: tail"><istex:xmlDeclaration>version="1.0" encoding="UTF-8"</istex:xmlDeclaration><istex:docType PUBLIC="-//ES//DTD journal article DTD version 4.5.2//EN//XML" URI="art452.dtd" name="istex:docType"></istex:docType><istex:document><converted-article version="4.5.2" docsubtype="sco" xml:lang="en"><item-info><jid>AJC</jid><aid>74902008</aid><ce:pii>0002-9149(74)90200-8</ce:pii><ce:doi>10.1016/0002-9149(74)90200-8</ce:doi><ce:copyright type="unknown" year="1974"></ce:copyright></item-info><head><ce:dochead><ce:textfn>Reports on therapy</ce:textfn></ce:dochead><ce:title>Control of tachyarrhythmias associated with Wolff-Parkinson-White syndrome by amiodarone hydrochloride</ce:title><ce:author-group><ce:author><ce:given-name>Mauricio B.</ce:given-name><ce:surname>Rosenbaum</ce:surname><ce:degrees>MD</ce:degrees><ce:cross-ref refid="COR1"><ce:sup loc="post">∗</ce:sup></ce:cross-ref></ce:author><ce:author><ce:given-name>Pablo A.</ce:given-name><ce:surname>Chiale</ce:surname><ce:degrees>MD</ce:degrees></ce:author><ce:author><ce:given-name>David</ce:given-name><ce:surname>Ryba</ce:surname><ce:degrees>MD</ce:degrees></ce:author><ce:author><ce:given-name>Marcelo V.</ce:given-name><ce:surname>Elizari</ce:surname><ce:degrees>MD, FACC</ce:degrees></ce:author><ce:affiliation><ce:textfn>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</ce:textfn></ce:affiliation><ce:correspondence id="COR1"><ce:label>∗</ce:label><ce:text>Address for reprints: Mauricio B. Rosenbaum, MD, Rivadavia 3820, P.B., A, Buenos Aires, Argentina.</ce:text></ce:correspondence></ce:author-group><ce:date-accepted day="9" month="1" year="1974"></ce:date-accepted><ce:abstract class="author"><ce:section-title>Abstract</ce:section-title><ce:abstract-sec><ce:simple-para view="all" id="simple-para.0010">Amiodarone hydrochloride proved to be highly effective in preventing and treating arrhythmias of the Wolff-Parkinson-White (WPW) syndrome in 11 patients with WPW conduction and recurrent tachyarrhythmias. Paroxysmal supraventricular tachycardia (six patients), atrial fibrillation (four patients) and atrial flutter (one patient) were the most significant arrhythmias. In most patients the arrhythmia was seriously disabling because of the extremely rapid ventricular rate, adverse hemodynamic consequences and frequent recurrence and long duration of the episodes. Other known antiarrhythmic agents were ineffective. In all 11 patients amiodarone, in doses of 300 to 600 mg daily, totally, easily and safety controlled the arrhythmias for periods of 2 to 8 months. The drug was fully effective after an average of 7 days of treatment. Tolerance to amiodarone was excellent. The occurrence of corneal microdeposits of the drug was the only Important undesirable effect, but subjective ocular disturbances were not noted. The microdeposits are reversible, and can be avoided by discontinuing the drug for 7 days every 1 to 2 months. Amiodarone apparently causes a significant prolongation of refractoriness in the normal (A-V node and His-Purkinje system) as well as in the anomalous pathway, thus creating favorable conditions for prevention and Interruption of any reentry mechanism requiring participation of both pathways.</ce:simple-para></ce:abstract-sec></ce:abstract></head></converted-article></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Control of tachyarrhythmias associated with Wolff-Parkinson-White syndrome by amiodarone hydrochloride</title></titleInfo><titleInfo type="alternative" lang="en" contentType="CDATA"><title>Control of tachyarrhythmias associated with Wolff-Parkinson-White syndrome by amiodarone hydrochloride</title></titleInfo><name type="personal"><namePart type="given">Mauricio B.</namePart><namePart type="family">Rosenbaum</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</affiliation><description>Address for reprints: Mauricio B. Rosenbaum, MD, Rivadavia 3820, P.B., A, Buenos Aires, Argentina.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Pablo A.</namePart><namePart type="family">Chiale</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">David</namePart><namePart type="family">Ryba</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Marcelo V.</namePart><namePart type="family">Elizari</namePart><namePart type="termsOfAddress">MD, FACC</namePart><affiliation>From the Services of Cardiology of Ramos Mejia Hospital, Salaberry Hospital and Argerich Hospital, Buenos Aires, Argentina</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="brief communication" displayLabel="Short communication"></genre><originInfo><publisher>ELSEVIER</publisher><dateIssued encoding="w3cdtf">1974</dateIssued><dateValid encoding="w3cdtf">1974-01-09</dateValid><copyrightDate encoding="w3cdtf">1974</copyrightDate></originInfo><language><languageTerm type="code" authority="iso639-2b">eng</languageTerm><languageTerm type="code" authority="rfc3066">en</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">Amiodarone hydrochloride proved to be highly effective in preventing and treating arrhythmias of the Wolff-Parkinson-White (WPW) syndrome in 11 patients with WPW conduction and recurrent tachyarrhythmias. Paroxysmal supraventricular tachycardia (six patients), atrial fibrillation (four patients) and atrial flutter (one patient) were the most significant arrhythmias. In most patients the arrhythmia was seriously disabling because of the extremely rapid ventricular rate, adverse hemodynamic consequences and frequent recurrence and long duration of the episodes. Other known antiarrhythmic agents were ineffective. In all 11 patients amiodarone, in doses of 300 to 600 mg daily, totally, easily and safety controlled the arrhythmias for periods of 2 to 8 months. The drug was fully effective after an average of 7 days of treatment. Tolerance to amiodarone was excellent. The occurrence of corneal microdeposits of the drug was the only Important undesirable effect, but subjective ocular disturbances were not noted. The microdeposits are reversible, and can be avoided by discontinuing the drug for 7 days every 1 to 2 months. Amiodarone apparently causes a significant prolongation of refractoriness in the normal (A-V node and His-Purkinje system) as well as in the anomalous pathway, thus creating favorable conditions for prevention and Interruption of any reentry mechanism requiring participation of both pathways.</abstract><note type="content">Section title: Reports on therapy</note><relatedItem type="host"><titleInfo><title>The American Journal of Cardiology</title></titleInfo><titleInfo type="abbreviated"><title>AJC</title></titleInfo><genre type="Journal">journal</genre><originInfo><dateIssued encoding="w3cdtf">197408</dateIssued></originInfo><identifier type="ISSN">0002-9149</identifier><identifier type="PII">S0002-9149(00)X0400-6</identifier><part><date>197408</date><detail type="volume"><number>34</number><caption>vol.</caption></detail><detail type="issue"><number>2</number><caption>no.</caption></detail><extent unit="issue pages"><start>A1</start><end>A2</end></extent><extent unit="issue 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